Szczepanik
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I think you're assuming too much in the timeline or sequence of how the body heals and presuming those milestones in recovery will be hit.
There is no guarantee the nerve reinnervates in length or to the extent to which pre-injury functionality can be regained. The channels/pathways the nerve follows as it regrows can atrophy and close up which can cause the "growing" nerve to reinnervate the wrong muscles. That's one issue. An entirely different issue is that the nerve can indeed reinnervate the right muscle, but not provide the same level of impulse that it once did pre-injury. Recovery from this injury is a matter of degree, not a simple it does or it doesn't situation.
For the average Joe, that can be a manageable minor impairment that allows him to walk with a hitch, but without the use of an AFO; he can mow the lawn, play catch with the kids, et cetera. For a pro athlete trying to perform at the highest level, it can be debilitating. Marcus Lattimore had a devastating knee injury that included nerve damage. He actually recovered but retired because he didn't feel he was going to be able to perform at the same level he was used to.
Atrophy shouldn't be an issue in an athlete with top notch medical care. If anything, they have been applying FES/TENS units to the area ( electrical stimulation units for those who don't know) , which can slow atrophy. It is extremely rare, and I have yet to hear of a case where a channel for a nerve closes up, even in people with spinal cord injuries, or other peripheral nerve injuries, I have not heard of that occuring often, if at all. I could be wrong though.
The loss of impulse you are referring to is the amplitude of a nerve, which only is affected if there is direct axonal damage. If there is no axonal damage, then there will be no amplitude change according to scientific literature. Nerve conduction speed (timing and activation) of a nerve is a different story, and that is most likely Jaylon's recovery hurdle.
The reason I assume much into the timeline of healing, is because I am currently going by the most recent scientific literature on timetables for neuron recovery. Which is always subject to change of course as science advances.
Prognosis from his injury is pretty heavily based upon the surgeon and the extent of which coverings of the nerve are damaged, depending on which tissues are damaged lead to pretty concrete prognostic factors.
The reinnervation of the incorrect muscles can occur, to my knowledge it is termed "collateral sprouting" , and occurs when there is axonal damage as well. It really depends on the severity of the nerve injury, but the surgeon should be able to easily identify whether or not itis a neuropraxia, axonotmesis, or neurotmesis.